Increased Urinary Urobilinogen Following Acute Myocardial Infarction

Increased urinary urobilinogen following acute myocardial infarction.

Increased urinary neopterin in acute myocardial infarction.

Kabadi et al. present additional data on patients with an established interference in the TDxJIM.x Total P4 assay (1, 2). This interference gives rise to abnormally low total T4 values in patients who have no apparent clinical thyroid abnormalities or laboratory results supporting a hypothyroid condition. The cause for this discrepant behavior has been attributed to an entity in patients’ sera ...

Rapid Diagnosis of Acute Myocardial Infarction

To improve the specificity of biochemical markers of myocardial infarction (MI) a method to measure cardiac troponin-I (CTn-I) was developed. CTn-I is a protein unique to cardiac muscle and is released after MI. Consecutive 150 patients admitted to the coronary care unit was studied. Value of CTn-I was determined in all samples. CTn-I concentration in the MI patients group was increased com...

The short-term survival following acute myocardial infarction: A prospective observational study

Introduction: To investigate 28 days survival rate following first acute myocardial infarction (AMI) associated with the presence of classical risk factors and treatment modalities in Gorgan, north of Iran. Materials and methods: Our cohort including all patients hospitalized due to AMI from 2010 to 2013. Data were collected on demographic, prophylactic drugs and classical coronary risk fa...

Acute Kidney Injury, Myocardial Infarction and Death Following Brake Fluid Poisoning; A Case Report

Background: Ethylene glycol is a toxic alcohol which is used in brake fluid, antifreeze, coolants, preservatives and chemical solvents. Ethylene glycol poisoning usually results in depression of the central nervous system, renal insufficiency and cardiopulmonary compromise, while laboratory findings include metabolic acidosis, increased anion gap, increased osmolar gap and calcium oxalate cryst...